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Acknowledgements The authors thank Brith Leidvik and Stefan Nordlander for technical and data assistance
There was a weak correlation between weight loss and SBP in the pooled analyses across SURPASS trials which further raises curiosity to explore potential weight-loss independent mechanisms that could be driving the reduction in SBP
Decades of peer-reviewed literature have characterised its role in redox homeostasis, glutathione-S-transferase-mediated xenobiotic conjugation, reactive-oxygen and reactive-nitrogen species buffering, hepatic detoxification biology, and protein S-glutathionylation as a redox-signalling post-translational modification
The consumption of fat reducers causes the impulses conditioning the intensive metabolic changes to reach the fat cells
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