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Studies in pre-clinical metabolic research models use the combination to examine the body composition and metabolic consequences of pharmacologically amplified GH pulsatility characterising GH-dependent lipolysis, lean mass accretion, and insulin sensitivity changes in response to secretagogue-driven GH axis stimulation

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However, tumors produce a variety of metabolic byproducts during growth, creating a tumor microenvironment (TME) that allows them to evade immune system clearance [256, 257]

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