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Instead, AOD 9604 is used to study downstream metabolic signaling, particularly adipose tissue lipolysis, allowing researchers to isolate lipid metabolism pathways without engaging the broader endocrine GHIGF-1 axis

This inflammatory state is coupled with NLRP3caspase-1 activation, oxidative/nitrosative stress and imbalances in lipid-derived (eicosanoids, bile acids, SCFAs) and amino acid-derived (glutamateGABA, tryptophanserotonin, NO) signalling pathways, all of which lower nociceptive thresholds

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