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Since abnormal lung regeneration and repair are potential processes in the pathogenesis of COPD, and the GHK-Cu complex plays a protective role in regeneration and wound healing, it is of significance to confirm whether GHK-Cu is effective in COPD as a protective agent against cigarette smoke-induced emphysematous destruction

Although one study emphasized genetic susceptibility while the other focused on molecular mechanisms, both point to SQOR as a core mitochondrial H 2 S-metabolizing enzyme that is critical for maintaining bone cell function and responding to oxidative stress