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One hypothesis is that we observed a greater induction of NRF2 through liver-specific Gclc deletion as compared to KEAP1 inhibition with CDDO-me because CDDO-me is a weaker inhibitor of NRF2-KEAP1 interaction compared to the ROS generated following GSH depletion

It is possible that the LKB1/AMPK/ACC pathway is activated by direct redox modulation of cellular proteins since activation of AMPK pathway has been directly linked to cellular redox status and hydrogen peroxide 49

Product Description Compound Specifications Full Name: Glycyl-L-Histidyl-L-Lysine Copper Peptide Abbreviation: GHK-Cu Sequence Type: Copper-binding tripeptide complex Amino Acid Sequence: GlyHisLys Molecular Formula: CHCuNO Molecular Weight: ~340.9 g/mol CAS Number: 89030-95-5 Structure Notes: Naturally occurring copper-binding peptide complex studied for its role in cellular communication, dermal signaling environments, and structural protein regulation Overview GHK-Cu is a naturally occurring copper-binding peptide complex frequently incorporated into experimental models examining how peptide-mediated signaling influences cellular communication , structural protein environments , and dermal signaling behavior